Huntingtin facilitates selective autophagy

Amir Gelman; Slobodkin, Moran Rawet Slobodkin; Zvulun Elazar

doi:10.1038/ncb3125

Huntingtin facilitates selective autophagy

Amir Gelman, Slobodkin, Moran Rawet Slobodkin, Zvulun Elazar^*

^*Corresponding author for this work

Department of Biomolecular Sciences

Research output: Contribution to journal › Short survey › peer-review

20 Citations (Scopus)

Abstract

Selective autophagy is essential for maintaining cellular homeostasis under different growth conditions. Huntingtin, mutated versions of which have been implicated in Huntington disease, is now shown to act as a scaffold protein that couples the induction of autophagy and the selective recruitment of cargo into autophagosomes.

Original language	English
Pages (from-to)	214-215
Number of pages	2
Journal	Nature Cell Biology
Volume	17
Issue number	3
Early online date	27 Feb 2015
DOIs	https://doi.org/10.1038/ncb3125
Publication status	Published - Mar 2015

All Science Journal Classification (ASJC) codes

Cell Biology

Access to Document

10.1038/ncb3125

Cite this

@article{3f565b8ea59b4ea8ab16154875e8c264,

title = "Huntingtin facilitates selective autophagy",

abstract = "Selective autophagy is essential for maintaining cellular homeostasis under different growth conditions. Huntingtin, mutated versions of which have been implicated in Huntington disease, is now shown to act as a scaffold protein that couples the induction of autophagy and the selective recruitment of cargo into autophagosomes.",

author = "Amir Gelman and {Rawet Slobodkin}, {Slobodkin, Moran} and Zvulun Elazar",

note = "NA",

year = "2015",

month = mar,

doi = "10.1038/ncb3125",

language = "English",

volume = "17",

pages = "214--215",

journal = "Nature Cell Biology",

issn = "1465-7392",

publisher = "Nature Publishing Group",

number = "3",

}

TY - JOUR

T1 - Huntingtin facilitates selective autophagy

AU - Gelman, Amir

AU - Rawet Slobodkin, Slobodkin, Moran

AU - Elazar, Zvulun

N1 - NA

PY - 2015/3

Y1 - 2015/3

N2 - Selective autophagy is essential for maintaining cellular homeostasis under different growth conditions. Huntingtin, mutated versions of which have been implicated in Huntington disease, is now shown to act as a scaffold protein that couples the induction of autophagy and the selective recruitment of cargo into autophagosomes.

AB - Selective autophagy is essential for maintaining cellular homeostasis under different growth conditions. Huntingtin, mutated versions of which have been implicated in Huntington disease, is now shown to act as a scaffold protein that couples the induction of autophagy and the selective recruitment of cargo into autophagosomes.

UR - http://www.scopus.com/inward/record.url?scp=84923810901&partnerID=8YFLogxK

U2 - 10.1038/ncb3125

DO - 10.1038/ncb3125

M3 - Short survey

SN - 1465-7392

VL - 17

SP - 214

EP - 215

JO - Nature Cell Biology

JF - Nature Cell Biology

IS - 3

ER -

Huntingtin facilitates selective autophagy

Abstract

All Science Journal Classification (ASJC) codes

Access to Document

Other files and links

Fingerprint

Cite this